Muscle Contraction and Plant Responses
Skeletal muscle fibres contain myofibrils made of repeating sarcomeres (Z line to Z line): thin actin and thick myosin filaments overlap. At the neuromuscular junction, ACh depolarises the sarcolemma; the impulse travels down T-tubules; the sarcoplasmic reticulum releases Ca²⁺. Ca²⁺ binds troponin, moving tropomyosin off the binding sites on actin. Myosin heads bind (cross-bridges), perform the power stroke (releasing ADP + Pᵢ), then ATP binds to detach them and is hydrolysed to recock the head. Actin slides over myosin: the I band and H zone shorten; the A band stays the same. Phosphocreatine regenerates ATP rapidly. Plants: auxin (IAA) causes cell elongation (acid growth) and phototropism; gibberellin causes stem elongation and stimulates amylase in seed germination; abscisic acid closes stomata in drought; the Venus fly trap closes by rapid changes in turgor after two hair touches.
Learning objectives
- Describe sarcomere structure.
- Explain the sliding filament model.
- Describe the neuromuscular junction.
- Describe plant responses controlled by hormones.
Why does rigor mortis happen after death?
Core idea
When you lift a bag, millions of protein filaments slide past each other, powered by ATP.
Calcium exposes binding sites on actin; myosin heads pull, release and recock using ATP. Plants respond with hormones instead of muscles.
Repeats while Ca²⁺ and ATP are present.
Myofibril 1.0 cm; sarcomere 2.5 µm.
- Goal
- Number.
- Strategy
- Convert units.
- 1 cm = 10⁴ µm.
- Divide.
Summary
- A band constant; I and H shorten.
- Ca²⁺, troponin, ATP.
- Auxin, gibberellin, ABA.
- Sarcomere
- Contractile unit between two Z lines.
- Auxin
- Plant hormone causing cell elongation.
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